Methylation and the folate cycle: MTHFR biochemistry and why folic acid may be toxic

Biochemistry and mechanisms of action

🧬 Methylation and the folate cycle: MTHFR biochemistry and why folic acid may be toxic

How one letter in a gene affects the absorption of the most popular vitamin B9

Methylation is one of the most fundamental biochemical processes in the body. It is the transfer of a methyl group (-CH3) to DNA, proteins and other molecules. It regulates gene expression, neurotransmitter synthesis, detoxification, cell repair and homocysteine metabolism. Methylation disturbances are linked to fatigue, mood problems, cardiovascular risks and developmental defects.

At the center of this process is the folate cycle, where the enzyme MTHFR (methylenetetrahydrofolate reductase) works. It is around MTHFR and synthetic folic acid that much controversy has arisen. Below is a full breakdown: history, benefits, food sources, norms and the role of active forms.

Key idea: by itself, MTHFR deficiency is not a "sentence diagnosis". MTHFR polymorphisms are very common (up to 30–40% of people). Problems arise when, against a background of reduced enzyme activity, a person gets a lot of synthetic folic acid, which is not converted into the active form and accumulates in the blood as UMFA.

📜 History of the discovery of folic acid

Folic acid was discovered in the 1930s–1940s in a search for a factor that treats anemia. This is a vivid example of how clinical observations led to a major discovery.

1

1931 — Wills' observation

Lucy Wills, a British doctor in India, noticed that pregnant women with anemia improved if they ate yeast or liver extract. Thus the "Wills factor" — future folate — was found.

2

1941 — isolation

Scientists isolated a substance from spinach, which they named "folic acid" (from Latin "folium" — leaf), because there is a lot of it in leafy greens.

3

1945 — chemical synthesis

American chemists synthesized folic acid artificially. This allowed its use in supplements and fortified foods.

4

1991 — prevention of neural tube defects

The MRC Vitamin Study proved: taking folic acid before conception reduces the risk of neural tube defects in the fetus. This led to mandatory fortification of flour with folates in dozens of countries.

Historical fact: fortification of flour with folic acid in the US and other countries led to a significant reduction in congenital neural tube defects — one of the successes of preventive medicine in the late 20th century.

⚙️ What is methylation and why it is needed

Methylation is a chemical reaction in which a methyl group is "attached" to a molecule. The main donor of methyl groups in the cell is SAMe (S-adenosylmethionine). It is formed from methionine and ATP, and after donating a methyl group it turns into homocysteine. So that homocysteine does not accumulate, it is converted back into methionine — and here folates and B12 are needed.

Roles of methylation: regulation of gene expression (epigenetics), synthesis of neurotransmitters (serotonin, dopamine, melatonin), detoxification, myelin formation, maintenance of DNA. A failure in this cycle affects many systems at once.
Donor

SAMe

Formed from methionine and ATP; donates methyl groups for hundreds of reactions, turning into homocysteine.

Return

Homocysteine recycling

Requires folates (B9), B12 and B6. This is exactly where MTHFR "works".

❤️ Why folates are good for humans

Folates (vitamin B9) are necessary for cell division and growth. Here are the main areas of their benefit:

Pregnancy

Fetal development

Folates are critically important in the first trimester for the formation of the fetal neural tube. Prevention of defects is the main reason for taking B9 before conception.

Blood formation

Red blood cell production

Folates are needed for blood cell division. Their deficiency causes megaloblastic anemia with fatigue and weakness.

Nervous system

Neurotransmitter metabolism

They participate in the synthesis of serotonin and other neurotransmitters. Deficiency is linked to low mood and "brain fog".

Heart and blood vessels

Lowering homocysteine

Folates together with B12 and B6 lower homocysteine levels — a risk factor for cardiovascular disease.

DNA

DNA synthesis and repair

Folates are a cofactor of nucleotide synthesis needed for copying and repairing DNA in dividing cells.

Information: folates are especially important for women of reproductive age, during pregnancy, with anemia, as well as people with elevated homocysteine and methylation disturbances.

🥗 Where folates are found: foods with numbers

Folates are found in large amounts in leafy greens, legumes and organ meats. Important: in food folates are present in natural form, while folic acid is the synthetic form in supplements and fortified products.

FoodFolates (mcg/100 g)Comment
Beef liver~200–300Record holder, + iron and B12
Spinach~190Leafy greens
Asparagus~150Good source
Broccoli~60–90Moderate
Brussels sprouts~60Moderate
Lentils~180Rich source
Chickpeas~170Rich source
Beans~130Good source
Avocado~80Moderate source
Beetroot~110Good source
Egg (1 pc)~22Moderate
Cooking tip: folates are sensitive to heat and light. Long boiling reduces their content (part goes into the water). It is better to eat leafy greens fresh, cook vegetables on steam or briefly stew. Legumes are an excellent and cheap source of folates in the diet.
Important: vitamin B9 from food is natural folates, which are absorbed more slowly and are better regulated by the body than large doses of synthetic folic acid from supplements. With high supplement doses, it is important to consider MTHFR polymorphisms.

💊 Folate norms per day and month

CategoryFolate norm/day
Adults400 mcg
Pregnancy600 mcg
Lactation500 mcg
Upper limit (from supplements)1000 mcg/day
Per month: at a norm of 400 mcg/day this is ~12,000 mcg (12 mg) of folates a month. It is quite realistic to cover this with food: a serving of lentils or spinach a few times a week + vegetables give a substantial part. But for the prevention of neural tube defects, doctors prescribe special supplement doses.
In pregnancy: for the prevention of neural tube defects, folic acid is taken 1–3 months before conception and in the first trimester in doses prescribed by a doctor (usually 400–800 mcg/day).

🧩 MTHFR, UMFA and active 5-MTHF

Synthetic folic acid must go through several enzymatic stages, including MTHFR, to become active. With reduced enzyme activity (polymorphism) or large doses, some folic acid remains in the blood unchanged — this is called UMFA.

Attention: a high level of UMFA raises researchers' questions — it is linked to a possible reduction in the activity of natural protective cells (NK cells) and is discussed in the context of risks. Therefore, when taking large doses of B9 and with MTHFR polymorphisms, active forms are often recommended.
C677T genotypeMTHFR activityComment
CC (normal)~100%Regular folic acid is absorbed normally
CT (heterozygote)~60–70%Regular intake is often enough, monitor homocysteine
TT (homozygote)~30–40%It is reasonable to consider the active form 5-MTHF
Regular folic acid

For most people

Cheap, proven effective for the prevention of neural tube defects. Requires the work of MTHFR.

Active 5-MTHF

With polymorphisms and high homocysteine

A ready active form, does not require MTHFR. Preferred with reduced enzyme activity.

Important: the presence of an MTHFR polymorphism does NOT automatically mean a need for supplements. Many people with the TT genotype live with normal homocysteine. The decision is made based on homocysteine analysis and the clinical picture, not on a single genetic test.
❓ Frequently asked questions
What is MTHFR in simple terms?

MTHFR is an enzyme that converts folates (vitamin B9) into the active form 5-MTHF, needed for methylation and lowering homocysteine. In some people its activity is reduced due to a polymorphism.

Which foods have the most folates?

Beef liver (~200–300 mcg/100 g), spinach (~190), lentils (~180), chickpeas (~170), asparagus (~150), beans (~130), beetroot (~110), broccoli (~60–90).

What is UMFA and why is it discussed?

UMFA is unmetabolized folic acid in the blood. It appears with high doses of synthetic folic acid, especially with reduced MTHFR activity. Its high level is discussed by researchers as undesirable.

How many folates do I need per day?

Adults 400 mcg/day, in pregnancy 600 mcg, in lactation 500 mcg. The upper limit from supplements is 1000 mcg/day.

Is folic acid really "toxic"?

Strictly speaking, no — it is not a poison. The term is exaggerated. It is about the accumulation of the inactive form UMFA with high doses and reduced enzyme activity, which may have undesirable effects.

Who is active 5-MTHF suitable for?

People with confirmed reduced MTHFR activity (homozygous C677T), elevated homocysteine, and as prescribed by a doctor. For most people, regular folic acid is effective.

What is homocysteine and why lower it?

Homocysteine is an amino acid, an intermediate product of methionine metabolism. An elevated level is linked to the risk of cardiovascular disease and cognitive impairment. It is lowered by folates, B12 and B6.

How to preserve folates when cooking?

Folates are sensitive to heat and light. Eat greens fresh, cook vegetables on steam or briefly stew, do not boil for a long time — part goes into the water.

How is L-methylfolate different from folic acid?

Folic acid is a synthetic precursor that requires activation by the enzyme MTHFR. L-methylfolate (5-MTHF) is already the active form, which works immediately, bypassing MTHFR.

Does MTHFR affect mood and fatigue?

Indirectly, yes. Methylation disturbances can affect neurotransmitter synthesis and general well-being, but the link is individual and requires evaluation with a doctor, not self-diagnosis by a gene.

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